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Evidenzbasierte Nahrungsergänzungsmittel ähnlich wie Tribulus, sortiert nach gemeinsamen Zielen und klinischer Evidenz. Vergleiche unten beliebige davon direkt miteinander.
A lipid-soluble antioxidant central to mitochondrial energy production, with the strongest trial support for fertility/IVF outcomes and heart failure.
Hormone-like vitamin that regulates 1,000+ genes — deficiency is widespread and linked to poor immunity, depression, and bone loss.
Nutritional insurance that fills micronutrient gaps. In the large COSMOS trial, a daily multivitamin improved memory and slowed cognitive decline in older adults — but it did not reduce total cancer, cardiovascular disease, or mortality.
Converts carbohydrates into energy and is needed for nerve function. Deficiency causes serious brain disorders, while extra thiamine is not shown to sharpen thinking.
Vitamin-like compound studied for hormonal and metabolic balance. Best evidence is for PCOS and gestational-diabetes prevention (multiple meta-analyses, though recent high-quality trials are mixed); anxiety/OCD data are older and thinner.
Bioactive folate that bypasses MTHFR conversion — critical for the 30-40% of people with gene variants affecting mood and methylation.
Adrenal hormone precursor that declines with age. In postmenopausal women with normal adrenal function, trials found no effect on sexual symptoms or metabolic markers.
A placental glycoprotein hormone that acts as a long-acting LH analogue: it binds the LH/CG receptor on testicular Leydig cells (and ovarian theca cells) to drive sex-steroid production. Honest appraisal: it is genuinely well-established for inducing ovulation/the final-maturation 'trigger' in female fertility, for inducing spermatogenesis in men with hypogonadotropic hypogonadism. Clinical use of hCG depends on the diagnosis and the exact product’s prescribing information. Evidence for adding hCG to testosterone treatment is more limited: a short study measured intratesticular testosterone, not pregnancy or live-birth outcomes. It does not establish that fertility will be preserved during testosterone therapy. Its proven benefits are confined to the gonadal axis — it does not improve systemic longevity or healthspan, and grey-market/compounded sourcing carries real risks.
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